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WILLIAM TASSMAN, MD; WILLIAM ANNESLEY, JR., MD

Arch Ophthalmol. 1966;75(5):608-614.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In the early 1950's supplementary oxygen for small premature infants was implicated as a contributing factor in retrolental fibroplasia (RLF). With this new knowledge available, subsequent efforts to restrict the use of oxygen for premature infants were made and a sharp drop in the incidence of RLF resulted. Since that time, however, new cases have continued to appear. Ten patients with severe retrolental fibroplasia which developed after 1958 were reported by a research group at the Massachusetts Eye and Ear Infirmary in January 1964.¹ In addition, many children who were premature at birth are now in school and have reached an age when vitreoretinal traction may cause retinal detachment. These patients have characteristic ocular changes which may include myopia and chorioretinal scarring. Because of vitreous syneresis and contraction of vitreous membranes, dragging of the retinal vessels and disc may also occur, as well as white without pressure, temporal retinal folds, . . . [Full Text PDF of this Article]

Author Affiliations
Philadelphia

From the Retina Service, Willis Eye Hospital, Philadelphia.

Footnotes
Submitted for publication Nov 26, 1965.

Read at the 17th Annual Eye Conference, Philadelphia, Feb 19, 1965.

Reprint requests to 626 Four Penn Center Plaza, 16th and Kennedy Blvd, Philadelphia 19103 (Dr. Tassman).

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