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FRANK J. MACRI, PhD

Arch Ophthalmol. 1965;73(4):528-539.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Introduction

Current findings strongly suggest that intraocular pressure (IOP) of the cat eye is dependent upon venous pressure^2-5,7-10 which in turn can be regulated by changes in resistance of the iris artery and its radial branches.⁶

Acetazolamide has been demonstrated to lower intraocular pressure, at least in part, by its ability to produce a fall in venous pressure^2,3 through its constrictive effect of the iris blood vessels.⁶ Many other agents, even though of diverse origin and activity, which have been shown capable of producing a lowering of IOP in patients with glaucoma, have all been found to have the common effect of producing an increase in resistance in the iris artery.⁹

Recently dilutions of serum were also shown capable of producing increases in resistance of the iris artery. Almost all of this serum vasoconstrictor activity could be ascribed to a substance which had many of . . . [Full Text PDF of this Article]

Author Affiliations
Bethesda, Md

From the Ophthalmology Branch, National Institute of Neurological Diseases and Blindness, National Institutes of Health, US Public Health Service, Department of Health, Education and Welfare.

Footnotes
Submitted for publication Oct 13, 1964.

Reprint request to Ophthalmology Branch, Nationaltute of Neurological Diseases and Blindness, Bethesda, Md. 20014.

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