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L. EUGENE WILLIAMS, BS; ANTHONY B. NESBURN, MD; HERBERT E. KAUFMAN, MD

Arch Ophthalmol. 1965;73(1):112-114.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Disciform keratitis is a term introduced by Fuchs in 1901 to describe a central corneal opacification that is seen most often as a late complication of herpes simplex keratitis.¹ Clinically, two kinds of stromal disease may occur after herpes simplex infection. In one type, dense, white, cheesy opacities which are generally eccentric occur. The pathogenesis of these lesions appears to be different from those of the typical disciform lesion. The disciform lesion is characterized by a round patch of corneal edema which is generally central and is typically surrounded by normal cornea. In the involved area of cornea, there are folds in Descemet's membrane, swelling of the stroma, and often epithelial bullae. In the early stages the disciform lesion is reversible, but as the edema persists, corneal scarring and permanent opacities often develop. Although the exact incidence of disciform keratitis following corneal herpes simplex is not known, in 1956, . . . [Full Text PDF of this Article]

Author Affiliations
Gainesville, Fla

Division of Ophthalmology, University of Florida College of Medicine.

Footnotes
Submitted for publication May 22, 1964.

This work was supported in part by United States Public Health Service grant No. NB 03538 from the National Institute of Neurological Diseases and Blindness of the National Institutes of Health.

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