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ROBERT P. THOMAS, MD

Arch Ophthalmol. 1964;72(4):529-534.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Introduction

The factors responsible for the precise control of the ocular pressure remain an enigma despite extensive research concerning the various neural, vascular, hormonal, humoral, and local mechanical influences. While much interest in the effect of ocular innervation upon intraocular pressure is apparent in the literature, no adequate demonstrations have been made of any sustained controlling influence.

The influence of the third cranial nerve and the parasympathetic outflow to the eye upon ocular hydrodynamics is somewhat confused despite the well-known effects of the various parasympathomimetic drugs. Schmerl and Steinberg (1949),¹ in one of a series of interesting experiments in rabbits, concluded that faradic stimulation in the region of the ciliary ganglion resulted, among other things, in an elevation in the ocular pressure. Perhaps more significantly, they noted a fall in the ocular pressure when destructive currents were applied in the same area. On occasion, this latter effect lasted for . . . [Full Text PDF of this Article]

Author Affiliations
Augusta, Ga

Medical College of Georgia, Division of Ophthalmology.

Footnotes
Submitted for publication April 7, 1964.

This work was supported in part by research grant No. NB 04218-02 from the National Institute of Neurological Diseases and Blindness of the National Institutes of Health, United States Public Service, and in part by the Medical Research Foundation of Georgia.

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