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An Electromyographic Study

ANDREW J. GAY, MD; JERRY BRODKEY, MD; JAMES E. MILLER, MD

Arch Ophthalmol. 1963;70(4):456-461.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Since Koerber¹ described the first case of retraction nystagmus in 1903, almost 50 others have been documented.² Pathologic studies seem to indicate that the responsible lesion is located in the periaqueductal region of the midbrain and perhaps in tectum or pretectum. An electromyographic study of patients with this abnormality was undertaken to gain some information concerning the peripheral mechanism which might offer indirect evidence related to central pathways.

Method and Patient Material

The apparatus and experimental method have been described in a previous publication.³ The following are abbreviated case histories of three patients with retraction and/or convergence nystagmus studied electromyographically.

Case 1

A 26-year-old white man noted the onset of double vision, frontal headache, nausea, and vomiting in March, 1961. Ventriculograms in May of that year demonstrated a block of the aqueduct, and a Torkeldsen procedure was done. Shortly thereafter, the patient developed a staphylococcal meningitis which . . . [Full Text PDF of this Article]

Author Affiliations
St. Louis

From the Department of Ophthalmology and the Oscar Johnson Institute, Washington University School of Medicine.; Present address: Jerry Brodkey, MD, Massachusetts General Hospital, Boston.

Footnotes
Submitted for publication March 8, 1963.

This investigation was supported in part by a research Grant, B-1349 from the National Institute of Neurological Diseases and Blindness of the National Institutes of Health, Public Health Service.

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