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ANDERS BILL, MD

Arch Ophthalmol. 1963;69(6):780-782.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The uveal venous pressure is difficult to measure in most species and, consequently, it has been a matter of considerable speculation and disagreement. Some investigators^7,8 assumed that under normal conditions the pressure in the proximal part of the uveal veins UVP, is just high enough to prevent collapse of the vessels, while others^9,10 claimed that it is considerably higher.

An increase in intraocular pressure, IOP, from the normal level, was considered by Kiss⁶ to produce such a compression of the narrow intrascleral part of the vortex veins as to cause a serious uveal venous stasis. Seidel,⁸ on the other hand, assumed that the UVP was raised just enough to balance the IOP.

In a previous paper¹ I demonstrated by indirect means that, in rabbits, the UVP is just above the IOP at normal and high IOP's and that the resistance in the veins draining the . . . [Full Text PDF of this Article]

Author Affiliations
Uppsala, Sweden

From the Institute of Physiology, University of Uppsala.

Footnotes
Submitted for publication Sept. 17, 1962.

This investigation was supported in part by grants from the Medical Faculty of Uppsala and in part by a grant (B-3060) from the National Institute of Neurological Diseases and Blindness, U.S. Public Health Service.

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