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SUMNER D. LIEBMAN, M.D.; ABRAHAM POLLEN, M.D.; STEVEN M. PODOS, A.B.

Arch Ophthalmol. 1962;68(1):72-78.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Traumatic hyphema is always potentially dangerous. Total hyphema ("eight-ball hemorrhage") has an especially poor prognosis because it often results in glaucoma, iris atrophy, and hematogenous pigmentation of the cornea.

The types of treatment advocated for traumatic hyphema include bed rest, sedatives, miotics, mydriatics, carbonic anhydrase inhibitors, and cold compresses. Some favor repeated paracenteses if the intraocular pressure rises.² Early paracentesis and injection of air have been advocated.³ When there is secondary bleeding and the anterior chamber becomes filled with blood, many operators advocate early surgery with irrigation and manual removal of clot.¹ Enzymatic dissolution of hyphema has also been attempted. Results with intramuscular trypsin are equivocal.^4,5

Jukofsky⁶ first suggested the use of streptokinase. When rabbit blood was injected into rabbit eyes with streptokinase, clots dissolved, but corneas became opaque. One total hyphema in a human patient cleared. Adverse reactions to streptokinase, including chemosis and iritis, . . . [Full Text PDF of this Article]

Author Affiliations
Boston

From the Massachusetts Eye and Ear Infirmary and the Harvard Medical School.

Footnotes
Submitted for publication Dec. 14, 1961.

This work was supported by the Edwin B. Dunphy Research Fund.

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