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ROBERT N. SHAFFER, M.D.; DANIEL I. WEISS, M.D.

Arch Ophthalmol. 1962;68(1):25-31.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Since 1905, when Heine described his neue glaucomoperation,¹⁴ cyclodialysis, this unique procedure has excited continuing interest. After a discussion concerning cyclodialysis and 1 of its complications, hypotony, we shall report on a case of long-standing postoperative hypotony (6 years) and the noteworthy result of therapy.

The mode of action of cyclodialysis has been diligently sought. It was Heine's contention that his operation created a pathway through which aqueous could reach the suprachoroidal space, where it would be absorbed. Elschnig¹⁰ and then Barkan³ histologically demonstrated the presence of this surgically created pathway or cleft in the successfully cyclodialyzed eye.

Some investigators^16,19 believe that cyclodialysis promotes the escape of aqueous through the usual exit channels either by facilitating access to, or egress from, the canal of Schlemm. By photographing the aqueous veins before and after transcorneal cyclodialysis, an admittedly crude technique, Saeteren and Thomassen²⁰ concluded that aqueous . . . [Full Text PDF of this Article]

Author Affiliations
San Francisco; New York

USPH Special Fellow in Ophthalmology, BT-823 (Dr. Weiss).; From the Department of Ophthalmology, University of California School of Medicine.

Footnotes
Submitted for publication Dec. 19, 1961.

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