 |
|
Aqueous and Blood Urea Nitrogen Levels After Intravenous Urea Administration
MILES A. GALIN, M.D.;
HUGO D. NANO, M.D.;
RICHARD A. DAVIDSON
Arch Ophthalmol. 1961;65(6):805-807.
|
|
| Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. |
|
|
|
The ability of intravenous urea to lower intraocular pressure has been well demonstrated.1 Though some authors considered a diuretic mechanism for its action, rather convincing evidence has been brought forth indicating the effect is due to the osmotic gradient established.2-5 It is known that this substance is maintained at a higher concentration in blood than in aqueous.6 However, it has not been determined what happens to this normal gradient at high blood levels of urea. It is the purpose of this communication to present the blood and aqueous urea levels following the intravenous administration of that substance to humans.
Materials and Methods
Patients hospitalized for routine cataract extraction were the subjects of this study. On the day of testing, venous blood was obtained for subsequent analysis of blood and plasma nitrogen. Shortly thereafter a 30% solution of lyophilized, ammonia-free urea* dissolved in 10% invert sugar was administered
. . . [Full Text PDF of this Article]
Author Affiliations
New York
Summer Student Fellow of The National Council to Combat Blindness (Mr. Davidson).; From the Department of Surgery (Ophthalmology) of the New York Hospital-Cornell Medical Center.
Footnotes
Submitted for publication Dec. 22, 1960.
Aided, in part, by grants from The National Society for the Prevention of Blindness, and The National Council to Combat Blindness.
Kindly supplied by Abbott Laboratories, North Chicago, Ill.
 CiteULike  Connotea  Del.icio.us  Digg  Reddit  Technorati  Twitter
What's this?
|
