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With an Electromyographic Study of Paretic Extraocular Muscles

R. O. SCHULTZ, M.D.; M. W. VAN ALLEN, M.D.; F. C. BLODI, M.D.

AMA Arch Ophthalmol. 1960;63(2):217-225.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Introduction

The pathogenesis of the ophthalmoplegia occurring in certain endocrine disorders has been subject to debate since Brain first coined the term "exophthalmic ophthalmoplegia" in 1938.¹ In his original description Brain¹ stated that thyrotoxicosis plays no essential role in ophthalmoplegia. In fact, the presence of impaired ocular motility associated with endocrine exophthalmos was thought to be evidence sufficient to rule out Graves' disease. However, in a recent review² of his last 50 cases of exophthalmic ophthalmoplegia he reported that 23 were thyrotoxic, 24 were euthyroid, and 3 hypothyroid. In all, 41 of the 50 either had had thyrotoxicosis or did have it at the time of examination.

This paper is a report of observations relating to external ophthalmoplegia and exophthalmos where these conditions have been associated with thyrotoxicosis. In addition, the relationship of ophthalmoplegia to proptosis is noted in patients without thyrotoxicosis who were considered to have . . . [Full Text PDF of this Article]

Author Affiliations
Iowa City

From the Departments of Ophthalmology and Neurology, State University of Iowa and the Sections of Ophthalmology and Neurology, Veterans Administration Hospital, Iowa City.

Footnotes
Submitted for publication Sept. 9, 1959.

Presented at the Midwestern Section of the Association for Research in Ophthalmology, Inc., Denver, April 18, 1959.

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