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ROBERT B. WELCH, M.D.; JACK C. COOPER, M.D.

AMA Arch Ophthalmol. 1958;59(5):665-675.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

This paper deals with the entities of macular edema, papilledema, and optic atrophy which occasionally follow apparently uneventful cataract surgery.

This discussion may be prefaced by noting that papilledema in association with ocular hypotony is a well-established clinical entity. Such changes have been observed following perforation of the globe, trauma to the eye without perforation, prolonged inflammation, and, especially, fistulizing operations for glaucoma. The importance of decreased intraocular pressure in the pathogenesis of this lesion is well established by many clinicopathologic and experimental studies.¹ Brégeat² has pointed out that in addition to papilledema there may be extension of edema into the macula which, if persistent, may result in macular cyst formation and degeneration. Thus it is well established that a variety of insults which produce hypotony may also be accompanied by the production of macular edema.

Reports of papilledema and macular edema following cataract surgery, however, have been . . . [Full Text PDF of this Article]

Author Affiliations
Baltimore

From the Wilmer Ophthalmological Institute of The Johns Hopkins Hospital and University.

Footnotes
Received for publication Sept. 12, 1957.

Presented at the Wilmer Resident's Association Meeting, April 4, 1957.

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