 |
|
Miosis in Argyll Robertson Syndrome and Related Pupillary Disorders
OTTO LOWENSTEIN, M.D.
AMA Arch Ophthalmol. 1956;55(3):356-370.
|
|
| Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. |
|
|
|
According to the classical description,* the Argyll Robertson syndrome is characterized by the following essential features: (1) the absence or diminution of the pupillary reflex to light; (2) an active or hyperactive pupillary contraction to near vision, and (3) the presence of miosis.
The cause of the miosis in the Argyll Robertson syndrome and in related pupillary disturbances has never been satisfactorily explained. In the abundant literature, extending over almost a century, two main theories have evolved:
- The miosis is due to an interruption of the sympathetic dilator fibers.
- The miosis is due to irritation within the parasympathetic reflex arc.
It is the purpose of this paper to discuss, on the basis of pupillographic and pharmacodynamic tests, the mechanism by which miosis is produced in the Argyll Robertson and related pupillary syndromes. An attempt will be made to clarify some of the basic problems which have perpetuated the controversies involved.
. . . [Full Text PDF of this Article]
Author Affiliations
New York
From the Department of Ophthalmology (Laboratory of Pupillography), Columbia University College of Physicians and Surgeons, and the Institute of Ophthalmology, Presbyterian Hospital.
Footnotes
Received for publication Nov. 30, 1955.
This work was supported by the Harriman Fund and by a grant from the U. S. Public Health Service (B-253 C2, Sens. Dis. 5).
 CiteULike  Connotea  Del.icio.us  Digg  Reddit  Technorati  Twitter
What's this?
|
