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PAUL A. CIBIS, M.D.; WERNER K. NOELL, M.D.; BERTRAM EICHEL, D.D.S.

AMA Arch Ophthalmol. 1955;53(5):651-663.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

One of the outstanding features of radiation damage is the difference between embryonic and adult tissues and their reactions and the manner in which these reactions are manifested (Hicks*; Russell⁴; Wilson and Karr⁵; Wilson, Jordan, and Brent,⁶ and Warkany and Schraffenberger⁷ ). The sequence of x-ray-induced developmental abnormalities and a strict timetable of their genesis have been established in rats and mice by Hicks.³ He demonstrated a certain resistance of the embryonic tissue toward x-ray doses in the range of 100-200 r during the first eight days of gestation. No measurable damage resulted, and the newborn rat appeared normal. Doses ranging from 300-400 r and administered to the rats during the first eight days either destroyed the embryos or resulted in normal newborn rats. The same doses applied at a later time led to definite developmental defects.

In Anderson's textbook of "Pathology,"⁸ the lymphoid tissues, . . . [Full Text PDF of this Article]

Author Affiliations
St. Louis; Buffalo; Randolph Field, Texas

From the Departments of Ophthalmology, Neuropsychiatry, and Pathology, U. S. A. F. School of Aviation Medicine, Randolph Field, Texas.

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