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ARTHUR H. KEENEY, M.D.; IRVING H. LEOPOLD, M.D.

AMA Arch Ophthalmol. 1952;47(6):720-727.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

FROM THE etiologic point of view, primary glaucoma continues to be one of the major enigmas of ophthalmology. One method of attacking the problem of etiology is to sift this disease complex for hidden, specific entities which render the ocular hypertension classifiable, in some cases, as secondary glaucoma. Work in this direction by Lindberg, in 1917, defined and separated the entity of glaucoma capsulare. Similarly, Ida Mann, in 1933, identified hyaline membranes of the anterior chamber, and subsequent studies by Reese and others removed this disease from the group of idiopathic glaucomas. Analogous efforts in the field of essential hypertension have removed the pheochromocytoma from that disease complex.

The purpose of this investigation is to search for possible faults in the general capillary mechanism which may be responsible for some cases of so-called primary glaucoma. Various, and often vague, concepts of a vascular genesis of glaucoma are nearly as old . . . [Full Text PDF of this Article]

Author Affiliations
LOUISVILLE, KY.; PHILADELPHIA

From the Research and Clinical Departments, Wills Hospital, Philadelphia.; Clinical Instructor in Ophthalmology, University of Louisville School of Medicine (Dr. Keeney); Director of Research, Wills Hospital, Philadelphia (Dr. Leopold).

Footnotes
This paper is a condensation of a thesis accepted by the University of Pennsylvania in partial fulfilment of the requirements for the degree of Master of Medical Science in Ophthalmology (Dr. Keeney).