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I. Morphological and Cytochemical Changes

LUDWIG VON SALLMANN, M.D.

AMA Arch Ophthalmol. 1951;45(2):149-164.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

SINCE it was first shown that roentgen ray cataract could be produced experimentally in mature animals (in guinea pigs by Bossuet,¹ in 1909, and in rabbits by Aulamo,² in 1928), the question of the primary site of radiation injury to the lens and the pathogenesis of the lesion have been discussed by many investigators on the basis of experimental and clinical evidence and have been studied systematically by a few. Most investigators have favored the concept of direct radiation damage to the lens (Bossuet,¹ Politzer,³ Rohrschneider,⁴ Peter,⁵ Grzedzielski,⁶ Okusawa,⁷ Leinfelder and Kerr,⁸ Goldmann and Liechti,⁹ von Szily,¹⁰ Krückmann,¹¹ Poppe¹²), whereas others, on the basis of the clinical characteristics of the cataract, have preferred the hypothesis of an indirect radiation effect induced by damage to the structures of the ciliary processes, with consequent nutritional disturbance of the lens . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the Department of Ophthalmology, Columbia University College of Physicians and Surgeons, and the Institute of Ophthalmology of the Presbyterian Hospital in the City of New York.

Footnotes
Read in part at the Eighty-Sixth Annual Meeting of the American Ophthalmological Society, Hot Springs, Va., June 1950.

The studies on the effect of roentgen rays on the lens, of which this paper is a part, are based on work performed under Contract #AT-30-Gen-70 of the Atomic Energy Commission.

This study was also supported by the Knapp Memorial Foundation.

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