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DAVID WEXLER, M.D.; GERALD BRANOWER, M.D.

Arch Ophthal. 1950;44(4):539-548.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

IN RECENT years attention has been focused on the study of the retinal capillaries in systemic vascular disease, since there is little question that the dynamic physiology of the retina is dependent on the integrity of the capillary circulation. In diabetes, for example, capillary microaneurysms have been described by all workers in this subject as the most characteristic retinal capillary lesion in this disease. In explanation of retinal hemorrhages and so-called exudates, such local vascular disturbances as capillary and precapillary stasis and dilatation were suggested by Elwyn¹ some years before retinal capillary lesions were observed histologically. Anatomically, Michaelson and Campbell² in 1940 pointed out that the deep retinal capillary plexus is a more closely knit meshwork than the superficial and that venous stasis occurs mostly in these deeper capillaries, where exudates and hemorrhages are more numerous.

For some years it has been recognized that in the study of . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the Laboratories and the Ophthalmological Division of the Mount Sinai Hospital.

Footnotes
Read before the New York Academy of Medicine, Section of Ophthalmology, March 20, 1950.

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