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IN RELATION TO PATHOGENESIS AND DIFFERENTIAL DIAGNOSIS

BERTHA A. KLIEN

Arch Ophthal. 1943;29(5):699-710.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The increasing number of reports during the past two years¹ of the results of heparin therapy in retinal venous occlusion should focus interest again on the various mechanisms by which this condition is produced, of which there are four. As will be shown, these four different types of pathologic change are by no means equally amenable to this anticoagulant therapy. In the present paper, special emphasis is placed on the differential diagnostic points which determine the pathogenesis of the occlusion and hence the success of therapy with heparin and dicoumarin (3,3'-methylene-bis[4-hydroxycoumarin] ).

The pathogenesis of the first and perhaps the most frequent type of occlusion of the central retinal vein, that on the basis of systemic angiosclerosis in older persons, has been elucidated by the investigations of Harms,² Scheerer,³ Verhoeff⁴ and Hertel.⁵ In the course of an essentially chronic sclerotic atrophic process, which affects the central . . . [Full Text PDF of this Article]

Author Affiliations
CHICAGO

From the Department of Ophthalmology of Rush Medical College.

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