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WARREN D. HORNER, M.D.

Arch Ophthal. 1936;16(3):447-461.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

An excellent review of the subject of di-nitrophenol in general was given in a report by the Council on Pharmacy and Chemistry of the American Medical Association,¹ from which I shall quote freely.

The actions of certain nitro derivatives of phenol and naphthol as accelerators of metabolism have been studied for the past fifty years. Cazeneuve and Lepine² in 1885 described the increase in the metabolism of dogs given di-nitronaphthol. A few years later Gibbs and Reichert³ reported similar observations with the use of di-nitrophenol.

During the war many cases of di-nitrophenol poisoning were observed among French munition workers. The poisoning, which was often severe and sometimes fatal, occurred so frequently that special pharmacologic study was instituted. The findings were reported on by Magne, Mayer and Plantefol⁴ in France and by Perkins⁵ in America.

In 1933 Tainter and Cutting¹ and their coinvestigators, proceeding independently, . . . [Full Text PDF of this Article]

Author Affiliations
SAN FRANCISCO

From the Department of Ophthalmology, the University of California Medical School.

Footnotes
Read before the Section on Ophthalmology at the Eighty-Seventh Annual Session of the American Medical Association, Kansas City, Mo., May 13, 1936.

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