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PHILLIPS THYGESON, M.D.; FRANCIS I. PROCTOR, M.D.

Arch Ophthal. 1935;13(6):1018-1021.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In previous reports¹ evidence was presented which indicated that trachoma is not a bacterial disease and which suggested that a filtrable virus is the etiologic agent. The nature of the Halberstaedter-Prowazek inclusion bodies,² found in epithelial scrapings from patients with trachoma, was discussed in a recent report;^1c on the basis of microscopic evidence, the conclusion was supported that the inclusion bodies of trachoma were intracellular colonies of a virus which might be the etiologic agent of the disease. The virus was found to be identical morphologically with the virus of inclusion blennorrhea³ and strikingly similar to the virus of psittacosis.⁴ Like these viruses it occurred in two phases: the large, initial body (Lindner) phase and the small, elementary body phase. By analogy it seemed logical to suppose that the elementary body phase would be the filtrable one. Experiments with suspensions of elementary bodies showed . . . [Full Text PDF of this Article]

Author Affiliations
IOWA CITY; SANTA FE, N. M.

From the Department of Ophthalmology, College of Medicine, State University of Iowa.

Footnotes
This study was aided by a grant from the Committee on Scientific Research of the American Medical Association.

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