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Michael Rauser, MD; Vivian Rismondo, MD
Baltimore, Md

Arch Ophthalmol. 1995;113(6):707-708.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Ischemic optic neuropathy is a common cause of vision loss in patients with giant cell arteritis (GCA). Systemic corticosteroid therapy has proved to be effective in preventing the ocular and systemic complications of giant cell arteritis, with loss of vision in long-term therapy being a rare occurrence.^1-3 We describe a patient with typical GCA in whom arteritic ischemic optic neuropathy developed after 3 weeks of steroid treatment, during which time the systemic signs and symptoms had responded to therapy.

Report of a Case.

A 74-year-old white woman noted a 2-week history of intermittent horizontal diplopia and a 1-month history of temporal headache and myalgia. She had lost 2.25 kg in the past week. Her medical and ocular history was unremarkable. The findings of the neuro-ophthalmological examination were normal (Figure 1), except for a mildly decreased visual acuity of 20/40 OU, which was consistent with cataracts. Findings of the laboratory . . . [Full Text PDF of this Article]

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