This Article  • References  • Full text PDF  • Reply to article  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

Gholam A. Peyman, MD
New Orleans, La

Arch Ophthalmol. 1995;113(6):701-702.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In the article in the August 1994 issue of the ARCHIVES, Guyer et al¹ reported on hyperpermeability of the choroid around the site of the dye leakage and elsewhere, undetectable clinically or by fluorescein angiography. They hypothesize that the pathogenesis of the central serous choroidopathy relates to abnormalities (hyperpermeability) of the choroidal vasculature.

Abnormalities of the choroid and their relationship to central serous choroidopathy could not be explained if permeability and the diffusion process across various structures of the retina and choroid had not been established. At the Jules Stein Eye Institute, Los Angeles, Calif, along with others, I evaluated the diffusion processes in the normal and photocoagulated retina, using peroxidase as a tracer material.^2-5 This information was significant in explaining excessive transudation in serous choroidopathy, accumulation of fluid under the sensory retina, and the effect of photocoagulation in treating this condition. Our studies demonstrated the following: (1) . . . [Full Text PDF of this Article]

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?