This Article  • References  • Full text PDF  • Reply to article  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citing articles on Web of Science (3)  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

Wilson C.-S. Wu, MD, PhD; Brian Mannion, MD, PhD; Richard M. Stone, MD
Boston, Mass

Arch Ophthalmol. 1995;113(4):408-409.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The immunologic mechanisms mediating intraocular inflammatory diseases are largely unknown. Certain cytokines have been implicated in the pathogenesis of uveitis. For example, interleukin-2 (IL-2) in ocular tissues¹ and IL-6 in aqueous^2,3 have been demonstrated in several forms of intraocular inflammation. However, it is unclear whether these molecules play a primary rather than secondary role in the pathophysiologic processes of uveitis.

We report a case in which therapy with IL-3 and IL-6 was associated with the development of bilateral uveitis and decreased visual acuity.

Report of a Case.

Anemia and thrombocytopenia due to myelodysplasia (subtype refractory anemia with excess blasts) that occurred 10 months after combination chemotherapy for nodular sclerosing Hodgkin's disease developed in a 67-year-old woman. At the time of enrollment into this trial, the patient was well, was without evidence of Hodgkin's disease or infection, had no other significant medical problem, and was receiving no medications other . . . [Full Text PDF of this Article]

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?