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David V. Pratt, MD; Silvia Orengo-Nania, MD; Barry L. Horowitz, MD; Osman Oram, MD
Houston, Tex

Arch Ophthalmol. 1995;113(2):141-142.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Oculomotor nuclear palsies are rarely observed without concomitant nonophthalmic neurological findings. We describe an isolated oculomotor nuclear palsy in a diabetic patient and correlate the spontaneous clinical improvement with radiologic images. Although the majority of diabetic oculomotor palsies may be peripheral and ischemic, we postulate they also can be central and hemorrhagic in nature.

Report of a Case.

A 60-year-old black woman with insulin-dependent diabetes, hypertension, and coronary artery disease presented with a sudden onset of horizontal diplopia. Ocular examination revealed bilateral ptosis that was worse on the left side, severe limitation of adduction, and a moderate decrease of infraduction and supraduction of the left eye. The right eye showed a mild decrease in supraduction secondary to a superior rectus paresis. A 30—prism diopter exotropia and a small right hypertropia were seen in the primary position. The findings of the rest of the neurological examination, including the pupil examination, . . . [Full Text PDF of this Article]

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