This Article  • References  • Full text PDF  • Reply to article  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

Stacia H. Goldey, MD; Latif M. Hamed, MD; Mark B. Sherwood, MD; Paul H. Phillips, MD
Gainesville, Fla

Arch Ophthalmol. 1992;110(12):1687-1688.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Glaucoma pituitary apoplexy is typically heralded by the onset of severe headaches along with a variable combination of impaired vision, ophthalmoplegia, and endocrinologic dysfunction.¹ We describe a patient who developed angle closure glaucoma presumably precipitated by mydriasis accompanying apoplexy-induced oculomotor palsy.

Report of a Case.

—A 47-year-old woman developed sudden severe headaches followed 3 hours later by diplopia. Examination revealed visual acuities of 20/200 in the right eye and counting fingers in the left eye. There were complete palsies of the left cranial nerves III, IV, and VI. Magnetic resonance imaging revealed a large mass within an expanded left hemisella, with erosion into the left sphenoid sinus and cavernous sinus (Figure). A presumptive diagnosis of pituitary apoplexy was made. Following transsphenoidal resection of the mass, the results of pathologic examination showed a necrotic, corticotropin-producing pituitary adenoma.

On the first postoperative day, a complete left ophthalmoplegia was again noted. The . . . [Full Text PDF of this Article]

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?