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Polymegethism of the Corneal Endothelium in an Eye With Long-standing Ptosis
Jan H. C. Kok, MD, PhD;
Erwin A. Dunnebier;
Carla P. Nieuwendaal, MD;
Aize Kijlstra, PhD
Amsterdam, the Netherlands
Arch Ophthalmol. 1992;110(11):1529-1530.
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| Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. |
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Because the corneal endothelium is the primary site for the metabolic pump that maintains corneal deturgescence, any change in the morphologic characteristics of endothelial cells is of considerable interest. It has been suggested that the functional reserve of the endothelium may be compromised with more severe cases of polymegethism.
Adverse effects of contact lens wear on physiologic features of the cornea were recently emphasized by the discovery that polymegethism and pleomorphism occurred with contact lens wear.1 These changes could be ascribed to oxygen shortage, carbon dioxide buildup, and pH changes under the lens.1 The same phenomena could be induced in the corneal endothelium of patients suffering from long-standing ptosis, which results in a decreased oxygen tension of approximately one third of the oxygen available under open-eye conditions.2
Report of a Case.
—We report the case of an 85-year-old woman with ptosis of the left eye for more than
. . . [Full Text PDF of this Article]
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