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Can Empty Sella Syndrome Be Mistaken for a Progressive Form of Nonarteritic Ischemic Optic Neuropathy?
Erkan Mutlukan, MD;
James F. Cullen, FRCS
Edinburgh, Scotland
Arch Ophthalmol. 1990;108(8):1066-1067.
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| Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. |
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To the Editor.
—We read with great interest the article by Sergott et al1 in the December 1989 issue of the ARCHIVES.
We find it difficult to determine the exact nature of their decompressive procedure. The authors persistently mention multiple linear incisions of the meningeal sheath and lysis dissection of arachnoidal adhesions within the subdural space, and they refer to the presence of cerebrospinal fluid and blood elsewhere within the subdural space. The subdural space is only a potential space and is normally empty, and frequent adhesions between dura and arachnoid are found in normal individuals.2,3 Cerebrospinal fluid is present and circulates in the subarachnoid space and it has been well established that an increase of pressure in this area is associated with papilledema, which can be relieved by decompression of the optic nerve sheath, but the arachnoid must be opened to achieve this goal. In the specimens
. . . [Full Text PDF of this Article]
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