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Gerald L. Wolf, MD
Christine Capuano

John Hartung, PhD
Brooklyn, NY

Arch Ophthalmol. 1985;103(9):1274.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In Reply.

—We did not make the "suggestion that in Abrams' cases N₂O anesthesia contributed to the loss of light perception on the first postoperative day." On the contrary, we restated Abrams' postulation that "N₂ entry into the SF₆ bubble, continuing for 24 hours, increased IOPs [intraocular pressures] and caused central retinal artery occlusion." Apparently, Merhige et al misread N₂ for N₂O. We did say that "if those patients had received 66% N₂O anesthesia, bubble volumes would have tripled and IOPs would have doubled during the operation....such increases might predict first postoperative day central retinal artery occlusion" [italics added], but that speculation does not appear to be contested.

This aside, we agree that reoperation with a short N₂O induction prior to switching to a halogenated anesthetic in O₂, along with central retinal artery monitoring and intraocular gas venting, . . . [Full Text PDF of this Article]

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