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Kenneth Merhige, MD; Harvey Lincoff, MD; Alan Van Poznak, MD
New York

Arch Ophthalmol. 1985;103(9):1272.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.

—The article by Wolf et al in the March 1985 ARCHIVES makes us aware of the danger of N₂O anesthesia in the presence of intraocular gas. Although we agree that N₂O anesthesia is potentially dangerous, we have found that it can be tolerated when used with adequate caution.

The suggestion that in Abrams' cases N₂O anesthesia contributed to the loss of light perception on the first postoperative day can only imply the unlikely possibility that occlusion of the artery occurred during surgery and was not recognized until the next day. Nitrous oxide anesthesia does not contribute to postoperative bubble expansion. Although N₂O enters the eye so rapidly that the volume of gas in a cat eye was shown to double in 30 minutes, its movement out of the eye is even more rapid when ventilation is used at the . . . [Full Text PDF of this Article]

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