This Article  • References  • Full text PDF  • Reply to article  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citing articles on HighWire  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

Martin Wand, MD
Hartford, Conn

Arch Ophthalmol. 1985;103(8):1112.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.

—It is now well accepted that there is a relationship between intracapsular cataract extraction/lensectomy and postoperative development of rubeosis iridis and neovascular glaucoma in patients undergoing vitrectomy.¹ Recently, my associates and I demonstrated the relationship between intracapsular cataract extraction and postoperative development of rubeosis iridis and neovascular glaucoma in diabetic patients, especially in patients with active proliferative diabetic retinopathy.² The article by Thompson and Glaser³ in the ARCHIVES is especially timely and significant because of the current trend toward extracapsular cataract extraction as well as the work being done in Glaser's laboratory on the isolation and identification of a diffusable angiogenesis factor.

Their article seemingly shows the importance of the posterior capsule of the lens as a barrier to the diffusion of 20,000- and 70,000-dalton dextran from the vitreous to the anterior chamber, thereby providing experimental data to possibly explain the above clinical findings. . . . [Full Text PDF of this Article]

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?