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Francesco Viola, MD; Giovanni Staurenghi, MD; Roberto Ratiglia, MD

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

We read with interest the article by Goldstein and colleagues in the November 2007 issue of the Archives about incidence and management of elevated intraocular pressure (IOP) in patients with uveitis treated with fluocinolone acetonide implants.¹ We would like to add a thought to the discussion of the findings.

They reported that 71% of implanted eyes developed elevated IOP requiring antiglaucomatous medication. Nevertheless, in 36.6% of implanted eyes IOP-lowering procedures were performed with a rate of hypotony of 42.5%. Forty-six percent of these eyes manifested hypotony more than 60 days following surgery and 18% more than 1 year after surgery. Thus we may conclude that hypotony occurred in 36% of implanted eyes right after IOP-lowering surgery. In these cases IOP-lowering surgery seems to be the cause of hypotony, rather than the ciliary body damaged by chronic inflammations or implant surgery.

We . . . [Full Text of this Article]

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RELATED ARTICLE

Intraocular Pressure in Patients With Uveitis Treated With Fluocinolone Acetonide Implants
Debra A. Goldstein, David G. Godfrey, Anthony Hall, David G. Callanan, Glenn J. Jaffe, P. Andrew Pearson, Dale W. Usner, and Timothy L. Comstock
Arch Ophthalmol. 2007;125(11):1478-1485.
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RELATED LETTER

Uveitis Treated With Fluocinolone Acetonide Implants—Reply
Debra A. Goldstein, David G. Godfrey, Anthony Hall, David G. Callanan, Glenn J. Jaffe, P. Andrew Pearson, Dale W. Usner, and Timothy L. Comstock
Arch Ophthalmol. 2009;127(1):116.
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