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Douglas A. Jabs, MD, MBA

Arch Ophthalmol. 2008;126(8):1143-1146.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

In 1981, a new disease, characterized by opportunistic infections (OIs) and unusual neoplasms, was reported to the Centers for Disease Control and Prevention.¹ This disease, AIDS, was soon thereafter discovered to be caused by the human immunodeficiency virus (HIV), which invades cells of the immune system, particularly CD4⁺ T cells, resulting in their loss and the subsequent immune deficiency. From the beginning, it was evident that the eye was a frequent target organ in AIDS.² The most frequent ocular manifestation was HIV retinopathy, consisting of cotton-wool spots with or without intraretinal hemorrhages.^2-3 Histologic and fluorescein angiographic studies also demonstrated the presence of other vascular abnormalities, including microaneurysms and telangiectatic vessels.^3-5 The most devastating ocular complications were ocular OIs, particularly cytomegalovirus (CMV) retinitis. Cytomegalovirus retinitis affected an estimated 30% of patients with AIDS sometime during the course of AIDS,⁶ and the rate among patients with CD4⁺ . . . [Full Text of this Article]

HIGHLY ACTIVE ANTIRETROVIRAL THERAPY


CMV RETINITIS IN THE HAART ERA

MANAGEMENT OF CMV RETINITIS IN THE HAART ERA

OTHER OCULAR COMPLICATIONS OF AIDS

AUTHOR INFORMATION


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