In reply
We thank Dr Hayreh for his interest and comment concerning our article.1
While proposing the hypothesis for the likely cause of visual loss in our patient, we considered the visual acuity (no light perception), the fundus picture (hyperemic disc edema and parapapillary hemorrhages), and the fluorescein angiographic characteristics (optic disc staining and "pruning" of perifoveal capillaries).
We agree with Dr Hayreh's assessment of transient occlusion, as was evident by normal arm-retina and arteriovenous transit times.
Visual acuity following CRAO is usually perception of hand motions or light perception, at least in parts of the visual field. The oxygen supply and nutrition from the choroidal vascular bed to the peripheral retina help in its longer survival and in maintenance of the peripheral visual field. Therefore, it is unlikely that the visual acuity of a patient with CRAO would be no light perception unless it is associated . . . [Full Text of this Article]
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Analogues