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Sohan Singh Hayreh, MD, MS, PhD, DSc

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

I was interested to read the case report by Singh and colleagues.¹ The authors state, "The likely cause of visual loss could be (1) ophthalmic artery occlusion with subsequent dislodge of fibrin emboli into the end arterioles at the posterior pole; or (2) retinal necrosis and macular infarction secondary to the aborted DIC (disseminated intravascular coagulopathy) process associated with toxic optic neuropathy (venom or ASV [antisnake venom] serum toxicity)." I am afraid that their diagnosis and explanation are not valid.

The eye had classic findings of transient central retinal artery occlusion (CRAO), as described in detail by us.^2-3 The fundus photograph and angiograms published by the authors¹ are identical to those published by us of transient CRAO² (see our² Figure 3). Thus, Singh and colleagues’¹ patient lost vision owing to transient CRAO caused by a transient embolism in the central retinal artery from . . . [Full Text of this Article]

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RELATED ARTICLE

Macular Infarction Following Viperine Snake Bite
Jatinder Singh, Preetam Singh, Rajbir Singh, and Vipin Kumar Vig
Arch Ophthalmol. 2007;125(10):1430-1431.
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RELATED LETTER

Transient Central Retinal Artery Occlusion Following Viperine Snake Bite—Reply
Jatinder Singh
Arch Ophthalmol. 2008;126(6):870-871.
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