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An Independent Risk Factor for Glaucoma?

Joseph Caprioli, MD

Arch Ophthalmol. 2007;125(8):1124-1125.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

Prospective, randomized, clinical trials of glaucoma treatment have convincingly shown the efficacy of intraocular pressure (IOP) reduction to slow the progression of glaucomatous optic nerve damage. Prospective, randomized, long-term studies have covered a spectrum of disease from early to late, and from normal to high pressures.^1-3 Others included a comparison of medical vs surgical treatment,⁴ and even treatment vs no treatment.⁵ All have demonstrated the strength of IOP reduction to slow the progression of disease, as measured by visual function and optic nerve structure. There is no longer any doubt (though some of us never doubted) that robust IOP reduction works in most patients.

A related issue is increasingly being debated. Is IOP fluctuation a separate and independent risk factor for glaucomatous damage? A few caveats are in order. Measurements of IOP may have the appearance of precision, but they are imprecise. Methods of measuring IOP differ, . . . [Full Text of this Article]

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