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Jatinder Singh, MS, DNB; Preetam Singh, DOMS; Rajbir Singh, MS; Vipin Kumar Vig, MS

Arch Ophthalmol. 2007;125(10):1430-1431.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

Macular infarction has been reported following toxic influences, for example, aminoglycoside toxicity.¹ Venomous snake bites may result in neurologic or hemostatic dysfunction. Viperine (hemotoxic) snake bites may produce coagulopathy, which may result in several systemic complications. Ocular involvement is rare. Common ocular problems encountered after a snake bite are generally neurologic (ptosis, ophthalmoplegia, accommodation paralysis, and optic neuritis). Visual loss may result from direct inoculation of venom into the eye (globe necrosis, keratomalacia, and uveitis), from optic neuritis, or secondary to hemostatic abnormality (vitreous hemorrhage, cortical infarction, and central retinal artery occlusion).^2-5

Report of a Case

A 17-year-old girl was bitten by a viperine snake. She was admitted to a local hospital in an unconscious state and administered first aid, anti–snake venom serum, and supportive care. She regained consciousness 14 hours after the snake bite and 6 hours later reported . . . [Full Text of this Article]

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Transient Central Retinal Artery Occlusion Following Viperine Snake Bite
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Transient Central Retinal Artery Occlusion Following Viperine Snake Bite—Reply
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THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES

Transient Central Retinal Artery Occlusion Following Viperine Snake Bite
Hayreh
Arch Ophthalmol 2008;126:870-871.
FULL TEXT  

Transient Central Retinal Artery Occlusion Following Viperine Snake Bite--Reply
Singh
Arch Ophthalmol 2008;126:870-871.
FULL TEXT