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Arch Ophthalmol. 2006;124:271-273.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

Congenital ectropion uveae (CEU) is a rare, nonprogressive condition scarcely mentioned in the ophthalmic literature. According to its etiology, ectropion is classified into 2 groups: acquired and congenital. It is believed that the cause of acquired ectropion is membranous traction of the iris from secondary causes such as inflammation and ischemia.¹

According to Dowling et al,² the term congenital ectropion was introduced in 1869 by Colsman³; however, it was later revealed that what was really described were flocculi. The first authors to actually describe congenital ectropion were Wicherkiewicz⁴ in 1891 and Spiro⁵ in 1896.

Congenital ectropion uveae consists of iris pigment epithelium on the anterior surface of the iris, anterior insertion of the iris, dysgenesis of the drainage angle, and glaucoma. Although the actual anomaly is nonprogressive, multiple studies^6-10 have linked it with the appearance of progressive open-angle glaucoma due to angle dysgenesis.

Most descriptions of CEU^{2, 6-9} have shown . . . [Full Text of this Article]

Report of a Case


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AUTHOR INFORMATION
Paul J. Harasymowycz, MD; Demosthenes G. Papamatheakis, MD; Ralph C. Eagle, Jr, MD; Richard P. Wilson, MD