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Arch Ophthalmol. 2003;121:1055-1056.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

Pigment dispersion syndrome and pigmentary glaucoma result from iridozonular friction causing disruption of the iris epithelium and deposition of iris pigment on anterior segment structures.¹ The classic triad of findings includes Krukenberg spindle, iris transillumination defects, and trabecular meshwork pigment. A posterior bowing of the iris that underlies the iridozonular contact and dispersion of pigment is often noted in these eyes. Usually seen in myopic patients, this concave iris configuration is believed to be due to reverse pupillary block, with the increased axial length allowing for a higher volume or pressure in the anterior chamber compared with the posterior chamber.^2-3 Laser iridotomy has been proposed as a therapeutic modality for pigment dispersion syndrome and pigmentary glaucoma by equalizing the pressure between the anterior and posterior chambers, in an analogous manner to the treatment of narrow-angle glaucoma, thereby eliminating the reverse pupillary block.² This results in a flat iris configuration with . . . [Full Text of this Article]

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Corresponding author and reprints: Paul M. Tesser, MD, PhD, Eye Surgeons and Physicians of St Louis Ltd, 224 S Woods Mill Rd, Suite 700, Chesterfield, MO 63017.