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Arch Ophthalmol. 2003;121:894-896.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

A pocket of fluid developing in the lamellar interface is a new complication that has previously been described in patients with steroid-induced intraocular hypertension after laser in situ keratomileusis (LASIK). The first case of interface fluid collection following LASIK was reported in 1999. Steroid-induced intraocular pressure (IOP) elevation and epithelial ingrowth were notable features in this case.¹ Subsequently, more cases of interface fluid and steroid-induced elevation of IOP have been reported.^2-5 The pathophysiologic mechanism behind the fluid collection in these cases is presumably the high IOP, which causes diffusion of aqueous humor across the endothelium into the interface, creating a fluid pocket in the lamellar interface. Clinically, this is usually associated with a fluid pocket visible on slitlamp examination in the lamellar interface, decreased visual acuity, a myopic shift in refraction, the presence of microcystic epithelial edema peripheral to the lamellar flap, an increase in central corneal thickness, steepening of . . . [Full Text of this Article]

Report of a Case


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Corresponding author and reprints: Daniel M. Albert, MD, MS, F4/338 Clinical Science Center, 600 Highland Ave, Madison, WI 53792-3220.

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