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A cohort of phakic patients with bilateral vitritis, cystoid macular edema, and retinal periphlebitis has been previously reported.¹ Despite long follow-up (an average of 13 years), these patients did not develop the typical characteristics of pars planitis, peripheral snowbanking, or multiple sclerosis. Additionally, these patients did not have a similar HLA antigen distribution as patients with known pars planitis.¹ We postulated that an inflammatory, infectious, or autoimmune insult resulted in these findings and examined the world literature for patients with similar findings.

In the Japanese literature, patients with similar funduscopic findings have been noted to have a high incidence of human T-cell lymphotrophic virus 1 (HTLV-1) infection.² Human T-cell lymphotrophic virus seropositivity was 26% in patients with idiopathic uveitis, 5% in an age-matched control population, and 3.4% in patients with known uveitic syndromes.³

We postulated that HTLV-1 and -2, although not endemic in Iowa, might be responsible for the disease . . . [Full Text of this Article]

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