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Arch Ophthalmol. 1998;116:1246-1248.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

Hemifacial atrophy (Parry-Romberg syndrome) is characterized by a slowly progressive atrophy of the skin, subcutaneous tissue, and muscle and bone of the face, usually beginning in the first 2 decades of life.¹ This disease progresses through an active phase of decompensation, generally lasting 2 to 10 years, followed by a quiescent phase without continued atrophy.² Ocular findings most commonly described include progressive enophthalmos, restrictive strabismus, pupillary disturbances, heterochromic iridocyclitis, and blepharoptosis.^{1, 3} To our knowledge, this case is the first report of primary corneal endothelial failure and penetrating keratoplasty in association with Parry-Romberg syndrome.

Report of a Case

A 59-year-old woman with a history of left hemifacial atrophy since age 15 years had continuous pain in the left eye that was unresponsive to topical lubricants. She had a history of ocular hypertension treated with a topical -blocker and a single episode of iritis in the left eye.

Ophthalmic examination revealed soft tissue atrophy of the . . . [Full Text of this Article]

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Corresponding author: Jerry G. Ford, MD, Wake Forest University Eye Center, Sixth Floor, Clinical Sciences Bldg, Medical Center Boulevard, Winston-Salem, NC 27157.