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Motilal Raichand, MD; Rutheva V. Dizon, MD; Krishan C. Nagpal, MD; Morton F. Goldberg, MD; Maurice F. Rabb, MD; Michael H. Goldbaum, MD

Arch Ophthalmol. 1978;96(9):1592-1596.

Abstract
• We report on four patients with sickle cell hemoglobin (SC) disease in whom macular holes developed. A major factor contributing to the formation of the holes was macular stretching, caused by simultaneous nasal traction from epipapillary fibroglial tissue and temporal traction from peripheral fibrovascular and fibroglial tissue. Another possible factor was macular ischemia.

Author Affiliations
From the Sickle Cell Eye Clinic, Comprehensive Sickle Cell Center, University of Illinois Eye and Ear Infirmary, Chicago.

Footnotes
Accepted for publication Nov 9, 1977.

Reprint requests to the University of Illinois Eye and Ear Infirmary, 1855 W Taylor St, Chicago, IL 60612 (Dr Goldberg).

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