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William E. Benson, BA; Emil Wirostko, MD; Harold F. Spalter, MD

Arch Ophthalmol. 1969;82(6):822-826.

Abstract
Simulated vitreous hemorrhages were created using labeled blood in order to study the effects of inflammation on the resorption of erythrocytes and on the residual vitreous opacification caused by blood elements. The inflammatory response of allergic uveitis and infection accelerates the removal of intravitreal blood. Unfortunately, attempts to utilize this information clinically to hasten clearing of the eye will not be possible because inflammation increases the ocular damage caused by iron and hemoglobin. Cortisone acetate, which decreases the inflammatory response to intraocular hemorrhage had no beneficial or harmful effect. Mercaptopurine, another anti-inflammatory agent, not only delayed the resorption of the blood, but increased the residual vitreous opacification.

Author Affiliations
New York

From the Department of Ophthalmology, Columbia College of Physicians and Surgeons, New York.

Footnotes
Submitted for publication April 3, 1969.

Reprint requests to Department of Ophthalmology, 635 W 165th St, New York 10032 (Dr. Spalter).

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